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Hanjoong Jo Hanjoong Jo, Ph.D.
Associate Chair for Emory Affairs
Professor
John and Jan Portman Professor in Biomedical Engineering

School of Medicine, Division of Cardiology
Emory University School of Medicine
Atlanta, GA 30322

Phone: 404-712-9654
Fax: 404-727-3330
Location: EU: HSRB - E 170
E-Mail: hanjoong.jo@bme.gatech.edu
URL: http://www.bme.gatech.edu/groups/jo

Google Scholar: CLICK HERE

Areas of Research

  • Mechanobiology and cardiovascular diseases, atherosclerosis and aortic valve disease
  • Role of shear stress in vascular inflammation and neovascularization
  • In vivo siRNA delivery

Educational Background

  • Juvenile Diabebetes Foundation Postdoctoral Fellow at Washington University, St. Louis and University of Alabama at Birmingham, 1999-2003
  • Ph.D., Pennsylvania State University, 1989
  • B.A., Korea University, 1984

Selected Awards

  • 2008, Journal of Molecular and Cellular Cardiology, Editorial Board Member
  • 2007, Cell and Molecular Bioengineering Journal, Associate Editor
  • 2007, Ada Lee and Pete Correll Professorship in Biomedical Engineering, Emory University
  • 2007, Outstanding Basic Science Research Award, Emory Medicine
  • 2006 - present, Am. J Physiology: Heart & Circ Physiology, Editorial Board Member
  • 2005 - present, Circulation Research, Editorial Board Member
  • 2004 - present, Am. J Physiology: Cell Physiology, Editorial Board Member
  • 1997 - 2001, NIH First Award
  • 1989 - 1991, The Juvenile Diabetes Foundation Postdoctoral Fellowship, New York, NY
  • 1983 - 1984, Lee, Myung-Hun Memorial Scholarship, Korea University
  • 1983, Presidential Award to the Most Outstanding Student, Korea University

Selected Research Publications

  • Tressel SL, Kim H, Ni C-W, Chang K, Velasquez-Castano JC, Taylor WR, Yoon Y-S, Jo H. Angiopoietin-2 Stimulates Blood Flow Recovery after Femoral Artery Occlusion by Inducing Inflammation and Arteriogenesis. Arterioscler Thromb Vasc Biol., in press, 2008.
  • Mowbray A, Rhee SG, Kang SW, and Jo H. Laminar shear stress upregulates peroxiredoxins (PRX) in endothelial cells - PRX 1 as a mechanosensitive antioxidant. J Biol Chem., 283(3): 1622-7, 2008.
  • Chang* K, Weiss D, Suo J, Vega JD, Giddens D, Taylor WR, and Jo H. Bone morphogenic protein (BMP) antagonists are co-expressed with BMP4 in endothelial cells exposed to unstable flow in vitro, in mouse aortas and in human coronary arteries- Role of BMP antagonists in inflammation and atherosclerosis. Circulation, 116: 1258-1266, 2007.
    *Accompanied by an Editorial. Circulation,116:1221, 2007.
  • Platt MO, Ankeny R, and Jo H. Laminar shear stress inhibits cathepsin L-dependent gelatinase and elastase activities in endothelial cells. Arterioscler Thromb Vasc Biol., 26: 1784-90, 2006.
  • Miriyala S, Nieto MCG, Harrison DG, Dikalov S, and Jo H. Bone morphogenic protein 4 induces hypertension by a mechanism involving endothelial dysfunction mediated by NADPH oxidases in C57BL/6 and ApoE-deficient mice. Circulation, 113:2818-2825, 2006.
  • Sorescu GP, Song, Tressel SL, Hwang J, Dikalov S, Smith DA, Boyd NL, Platt MO, Lassègue B, Griendling KK, and Jo H. Bone morphogenic protein 4 produced in endothelial cells by oscillatory shear stress induces monocyte adhesion by stimulating reactive oxygen species production from a nox1-based NADPH oxidase. Circ Res, 95:773-9, 2004.


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