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Areas of Research
- Mechanobiology and cardiovascular diseases, atherosclerosis and aortic valve disease
- Role of shear stress in vascular inflammation and neovascularization
- In vivo siRNA delivery
Educational Background
- Juvenile Diabebetes Foundation Postdoctoral Fellow at Washington University, St. Louis and University of Alabama at Birmingham, 1999-2003
- Ph.D., Pennsylvania State University, 1989
- B.A., Korea University, 1984
Selected Awards
- 2008, Journal of Molecular and Cellular Cardiology, Editorial Board Member
- 2007, Cell and Molecular Bioengineering Journal, Associate Editor
- 2007, Ada Lee and Pete Correll Professorship in Biomedical Engineering, Emory University
- 2007, Outstanding Basic Science Research Award, Emory Medicine
- 2006 - present, Am. J Physiology: Heart & Circ Physiology, Editorial Board Member
- 2005 - present, Circulation Research, Editorial Board Member
- 2004 - present, Am. J Physiology: Cell Physiology, Editorial Board Member
- 1997 - 2001, NIH First Award
- 1989 - 1991, The Juvenile Diabetes Foundation Postdoctoral Fellowship, New York, NY
- 1983 - 1984, Lee, Myung-Hun Memorial Scholarship, Korea University
- 1983, Presidential Award to the Most Outstanding Student, Korea University
Selected Research Publications
- Tressel SL, Kim H, Ni C-W, Chang K, Velasquez-Castano JC, Taylor WR, Yoon Y-S, Jo H. Angiopoietin-2 Stimulates Blood Flow Recovery after Femoral Artery Occlusion by Inducing Inflammation and Arteriogenesis. Arterioscler Thromb Vasc Biol., in press, 2008.
- Mowbray A, Rhee SG, Kang SW, and Jo H. Laminar shear stress upregulates peroxiredoxins (PRX) in endothelial cells - PRX 1 as a mechanosensitive antioxidant. J Biol Chem., 283(3): 1622-7, 2008.
- Chang* K, Weiss D, Suo J, Vega JD, Giddens D, Taylor WR, and Jo H. Bone morphogenic protein (BMP) antagonists are co-expressed with BMP4 in endothelial cells exposed to unstable flow in vitro, in mouse aortas and in human coronary arteries- Role of BMP antagonists in inflammation and atherosclerosis. Circulation, 116: 1258-1266, 2007.
*Accompanied by an Editorial. Circulation,116:1221, 2007.
- Platt MO, Ankeny R, and Jo H. Laminar shear stress inhibits cathepsin L-dependent gelatinase and elastase activities in endothelial cells. Arterioscler Thromb Vasc Biol., 26: 1784-90, 2006.
- Miriyala S, Nieto MCG, Harrison DG, Dikalov S, and Jo H. Bone morphogenic protein 4 induces hypertension by a mechanism involving endothelial dysfunction mediated by NADPH oxidases in C57BL/6 and ApoE-deficient mice. Circulation, 113:2818-2825, 2006.
- Sorescu GP, Song, Tressel SL, Hwang J, Dikalov S, Smith DA, Boyd NL, Platt MO, Lassègue B, Griendling KK, and Jo H. Bone morphogenic protein 4 produced in endothelial cells by oscillatory shear stress induces monocyte adhesion by stimulating reactive oxygen species production from a nox1-based NADPH oxidase. Circ Res, 95:773-9, 2004.
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